Boston University Creates COVID Strain With 80% Mortality In Mice

In an effort to research what makes Omicron so transmissible – and funded in part by grants from the NIH and Anthony Fauci’s NIAID, the researchers cobbled the Omicron spike protein to the original strain of Covid-19. The resulting virus was five times more infectious than Omicron.

Researchers at Boston University have created a new strain of Covid-19 that has an 80% kill rate in humanized mice.

“The Omicron spike (S) protein, with an unusually large number of mutations, is considered the major driver of these phenotypes. We generated chimeric recombinant SARS-CoV-2 encoding the S gene of Omicron in the backbone of an ancestral SARS-CoV-2 isolate and compared this virus with the naturally circulating Omicron variant,” reads the pre-print.

A nightmare in a lab has just taken place as we are grappling with the coronavirus’s evolving strains.
A COVID strain with an 80% death rate is said to have been created by Boston University researchers. In terms of severity, the Omicron virus and its variations are currently prevalent around the world. Health professionals have constantly cautioned us that the risk is still very high.
A group of researchers from Florida and Boston carried out the study at the university’s National Emerging Infectious Diseases Laboratories. This lab, one of 13 in the US with a biosafety level 4 designation, is working on risky research projects like those using COVID and Ebola.

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The team had made a hybrid virus — combining Omicron and the original Wuhan strain — that killed 80 percent of mice in a study

​What happened in the Boston University laboratory?​

The scientists genetically modified the primordial strain of coronavirus, which was first discovered in the wet market of Wuhan, China, using the spike protein that they isolated from the Omicron.
They documented how this hybrid, genetically modified strain reacted in mice.

​How lethal is the new virus?​

“In…mice, while Omicron causes mild, non-fatal infection, the Omicron S-carrying virus inflicts severe disease with a mortality rate of 80 percent,” they wrote in a research paper.
According to the researchers, the new strain has five times as many contagious virus particles as the Omicron version.
The virus hooks to the spike protein and enters human cells from there. The spike protein is becoming more potent with each successive variation, making it simpler for the virus to enter the host.

Laboratory made virus is a danger to mankind​

This has caused outrage to arise. After this disaster, people are now worried about their health and safety. Even some people question whether the Wuhan virus was accidentally created in a lab. It was thought that the coronavirus was created in the Wuhan Institute of Virology at the time the epidemic began.
‘This should be totally forbidden, it’s playing with fire,’ said Professor Shmuel Shapira, a leading scientist in the Israeli Government.
‘The research is a clear example of gain of function research,” Dr Richard Ebright, a chemist at Rutgers University in New Brunswick, New Jersey, told
“The government says law-abiding citizens can’t have a rifle or shotgun. They’re just too dangerous. Meanwhile, they’re literally engineering deadly viruses,” says Ezra Levant, a journalist.

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What does the University have to say?​

“First, this research is not gain-of-function research, meaning it did not amplify the Washington state SARS-COV-2 virus strain (original virus from 2020) or make it more dangerous,” BU said in a statement following online reports that the university called “false and inaccurate,” reported Boston Herald.
The discovery, according to the researchers, illuminates the pathogenic potential of the omicron.
Lead author Mohsan Saeed, defending their work and ignoring the criticism, told the Boston Herald that “this work shows that it is not the spike protein that drives Omicron pathogenicity, but rather other viral proteins. Determining those proteins will lead to better diagnostics and disease management strategies.”

Does this research serve its purpose?​

Yes, say the researchers.
80% of the mice perished after contracting the modified virus. However, all of the mice perished when they were exposed to the single strain of the ancestor virus.
According to the researchers, this analysis demonstrates that a number of other proteins are involved in the pathogenicity of Omicron, not the spike protein. According to main study author Mohsan Saeed, identification of such proteins will help develop better illness diagnostics and treatment plans.

How have authorities reacted to this?​

The National Institute of Allergy and Infectious Diseases which funded the project told STAT that Boston University’s original grant applications did not clarify that this specific work would be done. “The agency indicated it is going to be looking for some answers as to why it first learned of the work through media reports,” reported STAT.


by: Miss Cherry May Timbol – Independent Reporter
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100% Data Tampering